By Shahid M. Hussain MD, PhD (auth.)
This booklet offers a pragmatic strategy for MR imaging of the focal and diffuse liver lesions. it really is in line with state of the art MR imaging sequences, computer-generated drawings, concise determine captions, suitable and systematic (differential) diagnostic details, contemporary literature references, and sufferer administration probabilities.
MR imaging findings are correlated to ultrasound, computed tomography, and pathology whilst appropriate.
This ebook will drastically profit all execs and focused on imaging, prognosis, and remedy of focal and diffuse liver lesions, together with radiologists, gastroenterologists, surgeons, pathologists, MR physicists, MRI medical scientists, radiology and different citizens, MR technicians, and scholars.
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Extra resources for Liver MRI: Correlation with Other Imaging Modalities and Histopathology
Adam YG, Huvos AG, Fortner JG (1970) Giant hemangiomas of the liver. Ann Surg 172:239 – 245 10 Hemangioma III – Typical Giant 21 Fig. 1. Hemangioma, giant with a mid-size central scar, drawings. T2 fatsat: giant hemangioma is hyperintense to the liver with a brighter central scar (*); T1 in-phase: hemangioma is hypointense to the liver with a more darker central scar (*); ART: hemangioma shows a peripheral nodular enhancement; DEL: most of the giant hemangioma becomes enhanced except the central area including the central scar Fig.
Typical hemangiomas are hypointense on T1- and moderately hyperintense on T2-weighted images, and show peripheral nodular enhancement. The smaller the lesion the smaller the enhanced peripheral nodules will be. In the delayed phase, most hemangiomas show persistent enhancement as opposed to metastases, which show washout in delayed phase. Therefore, a combination of T2-weighted images and enhancement pattern is very specific, and allows characterization of even very small liver lesions (Figs. 3).
2). The brighter areas with the central scar most likely correspond to the myxoid tissue and the darker areas suggest the presence of fibrosis. In some cases, the central scar may even be larger than the periphery of the lesion. This suggests that the central scar may have been formed within the central vascular channels of the lesion after myxoid changes that eventually change into fibrosis. Despite this variability on T2-weighted images, the enhancement pattern should be quite characteristic on the dynamic gadolinium-enhanced images (Fig.