The second one revised variation of this article will replace and current present state-of-the-art scientific methods to this topic. This booklet will stay the resource textual content of data on drug-induced move problems authored and edited by means of the pioneers within the box. it will likely be a useful addition to the library of any neurologist.Content:
Chapter 1 stream issues: an outline (pages 3–19): Martin Cloutier and Anthony E. Lang
Chapter 2 ranking Scales for circulate issues (pages 20–29): Ikwunga Wonodi, Elliot L. Hong, Matthew T. Avila and Gunvant ok. Thaker
Chapter three Spontaneous circulate issues in Psychiatric sufferers (pages 30–52): Irene Richard, Christopher O'Brien and Roger Kurlan
Chapter four The Pharmacology of general and strange Antipsychotics (pages 55–71): Gary Remington and Shitij Kapur
Chapter five Acute Drug?Induced Dystonia (pages 72–102): Michael F. Mazurek and Patricia I. Rosebush
Chapter 6 Drug?Induced Parkinsonism (pages 103–139): Joseph H. Friedman, Martha E. Trieschmann and Hubert H. Fernandez
Chapter 7 Acute Drug?Induced Akathisia (pages 140–173): Lenard A. Adler, John Rotrosen and Burt Angrist
Chapter eight Neuroleptic Malignant Syndrome (pages 174–212): Stewart A. Factor
Chapter nine Tardive Dyskinesia (pages 213–256): Thomas M. Hyde, Jose A. Apud, Whitney C. Fisher and Michael F. Egan
Chapter 10 Neuroleptic?Induced Tardive Dyskinesia versions (pages 257–284): Frank Skidmore, William J. Weiner and Robert Burke
Chapter eleven circulate problems as a result of strange Antipsychotics (pages 285–301): Karen E. Anderson
Chapter 12 statement: Is Tardive Dyskinesia Disappearing? (pages 302–310): James B. Lohr
Chapter thirteen Dyskinesia triggered by way of Levodopa and Dopamine Agonists in sufferers with Parkinson's sickness (pages 313–350): John G. Nutt and Matthew A. Brodsky
Chapter 14 circulation problems and Dopaminomimetic Stimulant medicinal drugs (pages 351–370): Anthony E. Munson, Juan Sanchez?Ramos and William J. Weiner
Chapter 15 Antidepressants (pages 373–407): Maria L. Moro?de?Casillas and David E. Riley
Chapter sixteen Antiepileptics (pages 408–429): John C. Morgan and Madaline B. Harrison
Chapter 17 Miscellaneous Drug?Induced circulation issues (pages 430–441): Daniel Tarsy
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Additional info for Drug Induced Movement Disorders, Second Edition
There is a clear (but complex) interrelationship between OCD and tics. At least 17% of adults with OCD have tics, and increased rates of tics are reported in their relatives . Approximately 50% of patients with Tourette’s syndrome (TS) will show evidence of obsessive-compulsive symptoms [44, 45] and an increased prevalence of OCD is reported in first-degree relatives of TS patients independently of concurrent OCD in these probands . One family study on OCD has indicated that OCD is probably heterogeneous in origin, with familial forms, one tic-related, the other not, and a form that is neither tic-related nor familial .
Most studies of stereotypic behavior in experimental animals have emphasized the role of dopaminergic systems in the basal ganglia and limbic structures [5, 23]. Intrastriatal injection of dopamine and systemic administration of dopaminergic drugs, such as amphetamine or apomorphine, in rats produces dose-related stereotypic behavior [27–30]. These stereotypies can be prevented by pretreatment with dopamine receptor antagonist drugs . Studies indicate that the D2 dopamine receptor subtype mediates stereotypic behavior in animals and that activation of D1 receptors potentiates these D2-mediated effects [28–30].
Biochemical markers in the study of clinical effects and extrapyramidal side effects of neuroleptics. Psychiatry Res 1984; 13: 119–127. 15. Wojcik JD, Gelenberg AJ, LaBrie RA, et al. Prevalence of tardive dyskinesia in an outpatient population. Compr Psychiatry 1980; 21: 370–380. 16. Caligiuri MP, Bracha HS, Lohr JB. Asymmetry of neuroleptic-induced rigidity: development of quantitative methods and clinical correlates. Psychiatry Res 1989; 30: 275–284. 17. Hassin-Baer S, Sirota P, Korczyn AD, et al.