Disorders of the Prostate : New Concepts and Developments by Fouad K Habib

By Fouad K Habib

It has turn into transparent that medical discoveries and technological advances resulting in novel practices are taking place extra fast than they are often assessed via both medical trials or medical overview. still, very important breakthroughs were made within the improvement of recent remedies with major implications for the administration of benign prostatic hyperplasia and prostate melanoma. delivering a broad Read more...

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Identification of the genes differentially expressed between benign and malignant phenotype is only a small part of the work in searching for and determining the metastasisrelated genes. However, this part of the work is an essential first step, which may provide a pool of a large number of candidate genes for further analyzes, which will lead to the ultimate identification and isolation of the possible metastasis promoting- or suppressinggenes. Although many different methods are used to analyze differentially expressed genes, the majority of currently available techniques may be classified into two types: the hybridization-based techniques and PCRbased techniques.

Carboxyamidotriazole (CAT), an inhibitor of ROCC and arachidonic acid release, inhibits malignant proliferation and invasion of breast 02 Chapter 1194 27/4/04 9:27 am Page 34 34 The Prostate: New Concepts and Developments cancer (Lambert et al. 1997) and prostate cancer (Wasilenko et al. 1996). Phase II clinical trials for the treatment of androgen-independent prostate cancer are already occurring (Bauer et al. 1999). VGCC are structurally similar to voltagegated Naϩ channels (Tanabe et al. 1987).

1996), gene transcription (Berry & Nishizuka 1990) and apoptosis. Activation of PKC by phorbol esters has shown dose-dependent inhibition of cell proliferation and induction of apoptosis. Bryostatin-1, an alternative PKC activator, induces apoptosis with associated reduction of Bcl-2 expression and increased p53 protein expression (Mohammed, 1995). It has become apparent that individual PKC isoenzymes have discrete functions, specificity being generated by activation of co-factors, downregulation following activation, subcellular location and substrate specificity.

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